Thank you for clarifying that 2,4 D is not 2,4 Dinitrophenol. I learned
this from a medical forum from people that should have known better.
Now, why should we be concerned with any product that compromises ATP?
Granted the scientific studies, as designed and executed, do not point to a
problem. This may be due to several of the reasons we have stated on the
list already: a singe toxin being tested, the superiority of lab animal
diets, and tests of short duration. The web article below, identifying an
ATP depletion and recommending supplements to boost ATP production, is from
someone else's site. I had not even heard of fibromyalgia when I filed the
patent.
Based on this, we can argue any product that further compromises ATP in the
estimated 12,000,000 fibromyalgia sufferers should be further compelled to
prove its safety, not the other way around. ATP production is a
demonstrated problem in chronic fatigue syndrome and fibromyalgia and these
individuals are not aware of substances that can aggravate the condition.
Medicine is adhering to the theory that the body is attacking itself. It is
really rather a ridiculous theory and defies evolutionary theory as well as
the innate survival mechanism. Once one questions the validity of that
theory, one can explore other options freely, such as a search for an
external influence. The anti-phospholipid antibodies being produced then
become a survival mechanism to prevent certain factors from being tied up or
stored in cell membranes. It is a cellular choice--run at a suboptimal
level or die from the poison.
Can you identify this autoimmune disorder?
capillary rupture
bleeding gums
multiple spontaneous bruises
weakness
sore muscles in appendages
anemia
irregular heartbeat
difficult breathing
in children there may be bone abnormalities, failure to thrive
I can only imagine the attempts at finding the "problem" gene, the regimens
of steroids or chemotherapy, the support groups for sufferers that would
emerge as millions of dollars are poured into research to solve this
insidious disease where the body is obviously attacking itself. Thank
goodness the problem was solved before modern medicine got a hold of it:
scurvy
Detoxification
The substance that defines the priority of detoxification over rebuilding is
the high affinity, low capacity metabolic detoxifier phosphoadenosine
phosphosulfate (PAPS). Sulfate joins with ATP (and is probably the point
where the MSM contributes to pain relief) in two steps to form PAPS. At
this point PAPS can donate sulfur to proteoglycans and glycoaminoglycans
(rescued from obscurity and made famous by the book The Arthritis Cure).
These are components of cartilage, bone, skin, cornea, arteries, loose
connective tissue, joints, heart, and lungs to name of few. Basically, they
are ubiquitous in the body. PAPS, as tested in lab animals, has different
capacities just between the mouse and the rat. So, is it really accurate to
make human determinations from lab animals?
The sulfation process rids the body of toxins. However, it has a limited
capacity and competes for ATP and PAPS with other body processes. It is
here that we think that autoimmune disorders can have their origin as a
nutritional deficiency, as reversible as scurvy as long as there is no
permanent damage, and we can reduce the toxin and stress load (stress
depletes ATP) on the body.
Next
Part 2- Finding the flow of work in the machine we call the body
Part 3- Nutritionally reversing the fatal accelerated autoimmune disorders
in rhea (and ostrich and turkey): the data